Description
Masteron (Drostanolone Propionate) is one of the ‘exotic’ androgenic / anabolic steroids (AAS) that can be used by an athlete. It was originally developed in breast cancer treatment and used as an anti-estrogen (in the name of Masteril). SERM (Selective Estrogen Receptor Modulator) was used in combination with tamoxifen (Nolvadex) in the treatment of breast cancer and there was a significant decrease in estrogen levels in women exposed to such treatment. Today, it is not used for such purposes, for a variety of reasons, especially for athletes, including competing bodybuilders. Masteron, AS continues to be a favorite that does not like drugs.
The fact that Masteron has been used as an anti-estrogen continues to offer a great proposition about some of the features masteron has. Masteron is a derivative of DHT (dihydrotestosterone) and is not converted to estrogen by aromatization. It is contemplated that Masteron’s anti-estrogenic properties may be partly associated with an inhibition of either the aromatase enzyme or with estrogen interactions to block estrogen binding to the estrogen binding receptor. In both cases, Masteron will be a useful tool for many users who use estrogen-converting compounds (testosterone by many AS users, the main theme in many environments).By inhibiting the enzyme aromatase, Masteron will indeed block estrogen conversion through aromatization of the free testosterone. This solitary circulation agent does not increase marginally by removing the amount of free testosterone (thus testosterone leads to a greater effect on a system not containing Masteron) and the side effects of high estrogen levels, removing aromatization. Such side effects include gynecomastia development and water retention / swelling. Masteron, on the other hand, effectively inhibits binding to the estrogen receptor (ER) in an estrogenic state, limiting the ability of ER to bind estrogen when testosterone aromatization occurs.For this reason, with the help of this mechanism, the extreme effects of aromatizing production of estrogen will be limited to the use of Masteron. This removes aromatization. Such side effects include gynecomastia development and water retention / swelling. Masteron, on the other hand, effectively inhibits binding to the estrogen receptor (ER) in an estrogenic state, limiting the ability of ER to bind estrogen when testosterone aromatization occurs. For this reason, with the help of this mechanism, the extreme effects of aromatizing production of estrogen will be limited to the use of Masteron. This removes aromatization. Such side effects include gynecomastia development and water retention / swelling.Masteron, on the other hand, effectively inhibits binding to the estrogen receptor (ER) in an estrogenic state, limiting the ability of ER to bind estrogen when testosterone aromatization occurs. For this reason, with the help of this mechanism, the extreme effects of aromatizing production of estrogen will be limited to the use of Masteron.
Masteron thus has anti-estrogenic properties, but it has anabolic and androgenic properties (as DHT derivatives). Although it seems that there is no strong androgen on the theoretical and paper, Masteron has higher androgenic effects than expected. The use of Masteron as an AAS should not be considered as an option in post-course therapy because Masteron will prevent naturally occurring testosterone production and therefore cure because it has anti-estrogenic effects.
